Posted by ggggg123 on November 3, 2010, at 19:40:33
In reply to Re: To Cheryl-Lynne Anhedonia, posted by ggggg123 on November 2, 2010, at 14:52:38
It has been proven that reduced activity in the prefrontal cortex contributes to anhedonia, this is definately true in my case, as before I had anhedonia I used to have quite bad ocd, since my anhedonia I have no ocd ( ocd is caused by a high level of activity in the prefrontal cortex). SSRI's treat ocd, they reduce the activity in the prefrontal cortex and they therefore induce anhedonia, excitorary neurochemicals and lots of exercise are probably good strategies to try and regain that frontal lobe actvity. Basically anhedonia represents a frontal lobe syndrome, which is a common complaint of psychiatric meds and it would seem ssri's do play a big part in causing these symptoms and are not in anyway and advancement in psychopharmacology, only when an agent that restores normality, producing few side effects is released can we truly say that psychopharmacology has advanced, if anything its getting worse. If you have memory problems after taking an ssri, this is typical of the frontal lobe symptoms, people say ect is bad for memory, but at least you do not have the whole other array of side effects like the wonderfully brilliant ssri's.
Unfortunately by depleting NE/DA one will develop frontal lobe syndrome type symptoms and anhedonia, as neuronal activity is vastly reduced, it is inevitable. These chemicals fuel frontal lobe activity, without them sadly we become dawn of the dead extras.
poster:ggggg123
thread:10100
URL: http://www.dr-bob.org/babble/20101030/msgs/968268.html