Posted by psychobot5000 on January 24, 2009, at 15:23:09
In reply to Re: Agomelatine effects on OCD...(?), posted by desolationrower on January 23, 2009, at 12:49:44
> I'm not sure where tic activity originates; i think d2 antagonists are used for it though, so i guess i was wondering if the DA disinhibition from agometatine might be allowing it despite overall benefit...
> -d/rThat would seem to make sense, wouldn't it--a hard situation when I need more DA in the frontal cortex to help my depression and other issues...
Your hypothesis would make sense given that (I've read) antipsychotics may be the 'treatment of choice' for 'tic-related OCD.'
Maybe....theoretically it makes one think there could be a way of targeting the area of the brain responsible for OCD/Tics/whatever, and reducing activity there while leaving most of the rest functioning. But I imagine such a thing hasn't been invented yet (maybe deep-brain stimulation is the way to go for such relatively precise targeting...)
What you say makes good sense.
poster:psychobot5000
thread:869924
URL: http://www.dr-bob.org/babble/20090104/msgs/875877.html