> I remember reading that (in social phobia) there is an elevation of the dopamine transporter in the amygdala. Perhaps stimulants correct this.
>
> Interestingly, while I don't have social phobia per-se, I had lost much interest in social interaction. Basically interacting with others was difficult and boring. Since restarting methylphenidate, I find it much easier to connect with people, and my speech patterns are much more natural.
>
> Linkadge
So, does this mean (roughly) such an amygdala has an 'excess' of dopamine receptors, therefore the capacity to utilize dopamine is greater then what is naturally produced? (I don't mean to have you explain at length. My knowledge is limited.) Thanks.
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