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Re: hypercalcemia

Posted by jemma on April 4, 2003, at 11:54:09

In reply to Re: hypercalcemia, posted by Larry Hoover on April 3, 2003, at 18:26:38

> > This is interesting to me. I have ndi - nephrogenic diabetes insipidus - which has caused hypercalcuria and high blood phosphate, low sodium, etc. I control it with 25 mg. of hydrochlorthiozide. Because of ndi, I can't take lithium, which causes ndi in many people. So Mitch, your hypercalcemia may have been a result of lithium.
>
> Fascinating that a diuretic controls a disorder of diuresis. Have you ever been advised to use an NSAID? The combination is more effective than the thiazide alone.


I'll ask my endo about this - thanks.


>
> > Larry, how does any of this affect my meds? Currently I take 250 mg modafinil and 10 mg selegiline, along with the hydrochlorthiozide and HRT.
>
> None of these should be affected by your kidney disorder. Surely you were dose-titrated with foreknowledge of the NDI, yes?
>


Actually, I pretty much titrate the doses myself. After so many meds over the years, I've developed a pretty finely honed sense of what I need to feel well. And modafinil and low-dose selegiline aren't meds that my psychopharmacologist has much experience with.

> >I've often wondered if my brain fox came from a lack of vasopressin receptors in my hypothalamus, as well as my kidney.
>
> Were your psychotropic meds prescribed to deal with brain fog, in particular? They seem to be appropriate for that.

I found these meds and sold my pdoc on them. But yes, my depression is of the anhedonic, anergic, brain fog variety. Ssri's made things worse - after nine years on zoloft I discovered it had been downloading catecholamine receptors. Serzone turned me into a blithering idiot, and lamictal wasn't much better. Even wellbutrin made me fall asleep. Finally we tried modafinil, and voila - energy and motivation for the first time in a decade. Then he tried ritalin and it felt like valium, but with a wicked rebound effect. So I talked him into the selegiline, and its working a treat.

>
> As I understand it, the hypothalamus senses solute concentrations directly, and thus mediates vasopressin release, rather than responding to it. Or have I got that mixed up?

Like most things neurochemical, the V2 receptors seems to be pretty much a mystery. I do know that vasopressin not only prevents diuresis but also sharpens the mind and memory brilliantly.

>
> > My ndi appears to be genetic, but it's labelled idiopathic for 'who knows?'
>
> Ya. I hear ya. If experts don't know, who does?
>
> >I've had a lot of weird things go wrong, including postcapsular cataracts. I'm 47, and not diabetic.
>
> Well, I hate to say it, but you're of an age where things, idiopathic things, tend to become prominent.

Don't I know it. Like a car, wearing out its parts one by one. Damn, I wish I'd spent more time on early maintenance :^). Still, I have a lot of pretty weird things going on.

>
> > Feel free to ignore this. But I've come to admire the scope of your knowledge of the brain's and body's chemistry.
> >
> > - jemma
>
> I will always be a student. I have an infinite curiosity. Glad to find that suits others, too.
> ;-)
>
> Lar

Lucky for us. Thanks, Larry.

- jemma


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poster:jemma thread:215138
URL: http://www.dr-bob.org/babble/20030402/msgs/216192.html